The number of infected people does not reveal the level of spread of the pathogen at the planetary level. It is possible that the pathogen, if persistent or chronic infections, could be hiding in its genomic state in long-lived neurons, or in ganglia, a collection of neuronal bodies, which could be linked to the activity of neurons. Therefore, the virus may proliferate in conjunction with the activity of the neurons. However, the mechanism of the pathogen’s growth is completely unknown. Therefore, if PCR and other tests are repeatedly positive and negative over the course of a day, the actual number of people infected cannot be known with current science. As for the number of deaths, there is no way to determine the cause of death because in many cases the cause of death has not been investigated, so there is no other way but to look at the statistics compiled by the United Nations. However, since the death statistics of infected people have been measured at some level, we decided to look at how the statistics have changed. This is an interim measure to measure the level of planetary invasion. Right now, it’s the only way to go.https://www.aljazeera.com/news/
The currently spreading coronavirus is said to be a G strain that is more infectious than the D strain that first occurred in Wuhan City, Hubei Province, China. The G strain is already spreading in Australia. Possibility of strong infectivity It was concluded that it was able to spread farther faster and faster than the first strain, as it was more infectious and spread than the first strain. It was so dramatic in the early days of the global epidemic. It’s amazing that SARS-CoV-2 strains were able to make an evolutionary evolution
In a very contradictory view, this pathogen can be a persistent chronic infection that causes the genome to latent in a gel within the cell.
Since the mutation of the gene sequence in the genome of this pathogen suppresses the immune system in some way, it is considered that the frequency of occurrence tends to be extremely high.
It is concluded that the changes in the three-dimensional structure of the spike protein, which serves as an antigen for the antibody, vary very significantly.
There seems to be individual differences in the incubation period, or incubation time.
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